Hoffer A: The Schizophrenia, Stressand Adrenochrome Hypothesis.

Seven loci and a variety of phenotypes are now known(Neurology 72: 410, 2009).

This may be an example ofmolecular mimicry (Nat.

There is evidence that environmental toxins play a part in poor school performance. For example, environmental polycyclic aromatic hydrocarbons (PAH) at levels recently encountered in New York City air are thought to adversely affect children’s cognitive development at three years of age.

One known example of a blow to the unbornchild causing porencephaly: Dev.

The Controllers: A New Hypothesis of Alien Abduction

Immig K, Gericke M, Menzel F, Merz F, Krueger M, Schiefenhövel F, Lösche A, Jäger K, Hanisch UK, Biber K, Bechmann I. CD11c-positive cells from brain, spleen, lung, and liver exhibit site-specific
immune phenotypes and plastically adapt to new environments. Glia. 2015 Apr;63(4):611-25.

The best example is atrophy of the lateral geniculate body in people who havehad an eye enuclated.

More fascinating is the finding that these mutations may be inherited by children. Environmental toxins have been shown to alter the activity of genes through at least four generations after exposure. For example, women who smoke while pregnant double the risk of asthma in their grandchildren.

Waltrip RW 2nd, et al., Borna disease virus and schizophrenia. . 1995 Jan 31; 56(1):33-44.


The Synaptic Hypothesis of Schizophrenia: Neuron

163: 1404, 2006; and the lawyers areall over this; it's emerged that during a six-month period, about 20% of those collectingwelfare for mental illness will admit to hurting someone or at leastthreatening someone with a lethal weapon; same rate as the Duke study above.)The truth is that schizophrenics are more likely than non-schizophrenicsto commit all categories of crime except sex crimes (Lancet 355:614, 2000).

The Synaptic Hypothesis of Schizophrenia

There are three major causes of hypoglycemia – injections of insulin, organic pathologies, and unknown causes. Organic hypoglycemias can be endocrine-related, including problems in the pituitary, adrenal, or thyroid insufficiency. Changes in the pancreas may also occur. Autoimmune disorders , such as Lupus, Sjögren Syndrome, Hashimoto Thyroiditis, Addison’s Disease, rheumatoid arthritis, Type 1 diabetes, polymyositis (muscle inflammation), scleroderma (a connective tissue disorder), vitiligo (loss of skin pigment), and pernicious anemia (low B12 levels) also play a part in blood sugar stabilization. It is the unknown causes group that is most frequently diagnosed as mental illness.

cell signaling, and the “two-hit” hypothesis of schizophrenia

Today's historians say a generation of mothers needsan apology: Lancet 379: 1292, 2012.) In the early 1970's, it was all the fault of "the schizophrenogenic father"instead; as with the "S-mother", evidence was "anecdotal" (i.e., the biological parents of crazy people act screwy themselves; stress sometimes precipitates symptoms) and "based on sound theory" (i.e., "all people are born the same", the left-wing/Skinnerian ideology of the day).

the basis of the ‘two hit hypothesis’

Schizophrenics consume 2.5% of health care expenditures, constitute 10% of the totally and permanently disabled, and represent around 14% of the homeless (Psych.

• Proposes the two-hit hypothesis of schizophrenia.

Moreover, different types of olfactory deficits are found in different pathologies. For example, people with schizophrenic disorders often have normal odor sensitivity, but exhibit significant deficits in odor identification, recognition, and discrimination. Those suffering from depression will often exhibit deficits in the hedonic aspects of olfaction, even if, in some cases, alterations in sensitivity or identification are also found. In other words, they lose the pleasure of olfaction. Olfactory hallucinations in depression are often those of foul odors. Interestingly, two-thirds of these people believe the odor to emit from their own bodies. A 2008 study by J.F. Dileo showed that in soldiers in PTSD olfactory identification deficits as a predictor of aggression and impulsivity.